Periodontics · patient case with 2 questions

Painful gums and a foul odour

Patient: Male, 24 years old

Chief Complaint: “My gums are killing me and my breath is terrible.”

Background: University finals week. Smokes half a pack a day, sleeping four hours a night, eating poorly. Reports a sore throat two weeks ago.

Vital Signs: BP 124/78 mmHg · HR 88 bpm · T 99.6°F

Findings: Cratered, punched-out interdental papillae in the mandibular anterior region covered by a greyish pseudomembrane. Spontaneous bleeding on gentle retraction. Marked fetor oris. Submandibular lymphadenopathy. Generalised plaque and calculus; dry mucosa.

Question 1

What is the most likely diagnosis?

  1. Primary herpetic gingivostomatitis
  2. Plaque-induced chronic gingivitis
  3. Necrotising ulcerative gingivitis
  4. Desquamative gingivitis
Show answer and explanation

Correct answer: C. Necrotising ulcerative gingivitis

  • A. Primary herpetic gingivostomatitis is a childhood illness with high fever, malaise and vesicles that ulcerate across ALL mucosal surfaces, including non-keratinised tissue. It does not selectively crater the papillae.
  • B. Plaque-induced gingivitis gives erythema, oedema and bleeding on probing, but no necrosis, no pseudomembrane and no characteristic odour.
  • C. CORRECT. The diagnostic triad is PAIN, spontaneous BLEEDING and NECROSIS of the interdental papillae, which is what gives the cratered or punched-out appearance. Add the grey pseudomembrane, the distinctive fetid odour and the classic predisposing background — stress, smoking, sleep deprivation, poor nutrition — and the diagnosis is necrotising ulcerative gingivitis. Fusiform bacteria and spirochaetes predominate.
  • D. Desquamative gingivitis is a clinical description, not a diagnosis — diffuse erythema with epithelial sloughing, usually from lichen planus or mucous membrane pemphigoid. There is no papillary necrosis or fetor.

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Question 2

Each of the following is contributing to this patient's halitosis EXCEPT one. Which is the EXCEPTION?

  1. Poor oral hygiene with tongue coating
  2. Reduced salivary flow
  3. The calculus deposits themselves
  4. The necrotising gingival lesions
Show answer and explanation

Correct answer: C. The calculus deposits themselves

  • A. The posterior dorsum of the tongue is the single largest reservoir of odour-producing anaerobes in the mouth; a coated tongue with poor hygiene is the commonest cause of halitosis overall.
  • B. Saliva clears bacteria and substrate and buffers the mouth. When flow drops, anaerobic proteolysis accelerates and volatile sulphur compounds accumulate — the mechanism behind morning breath.
  • C. CORRECT as the exception. Calculus is a mineralised, essentially inert deposit. It contributes to halitosis only INDIRECTLY, by providing a rough retentive surface for the living biofilm that actually produces the odour. The calculus itself is not a source of volatile sulphur compounds.
  • D. The necrotic tissue in NUG is a potent direct source of volatile sulphur compounds — hydrogen sulphide and methyl mercaptan — which is why the odour of NUG is so characteristic that experienced clinicians recognise it before looking.

Note: The recall listed the options confusingly and appended a separate note about calculus. Reconstructed here as the version the examiner was most likely testing: calculus is a retentive factor, not a direct odour source.

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