Endodontics · patient case with 6 questions
Full-Mouth Pulpal Diagnosis
demo: 46-year-old female
chief: “Several of my teeth on the upper right have been bothering me in different ways.”
history: Non-contributory. No medications.
meds: None
findings: Tooth #2: no response to cold, painful to percussion and palpation, periapical radiolucency. Tooth #3: sharp pain to cold that lingers more than 20 seconds. Tooth #4: exaggerated but non-lingering response to cold, deep restoration. Tooth #5: pain on biting, tender to percussion, widened PDL at the apex.
Question 1
What is the pulpal and periapical diagnosis for tooth #3?
Show answer and explanation
Correct answer: C. Symptomatic irreversible pulpitis
- A. The pulpal response is clearly abnormal, so the pulp is not normal.
- B. A necrotic pulp gives no response to cold at all.
- C. Correct. A thermal response that lingers beyond about 10–20 seconds signals inflammation that will not resolve — symptomatic irreversible pulpitis. Root canal therapy or extraction is indicated.
- D. Reversible pulpitis produces a sharp response that disappears as soon as the stimulus is removed.
Question 2
What is the diagnosis for tooth #2?
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Correct answer: B. Pulp necrosis with symptomatic apical periodontitis
- A. There is no history or radiographic evidence of previous endodontic treatment.
- B. Correct. No response to cold indicates necrosis; pain on percussion and palpation with a periapical radiolucency indicates symptomatic apical periodontitis.
- C. Irreversible pulpitis requires a vital, responsive pulp — this pulp does not respond.
- D. Reversible pulpitis cannot coexist with a non-responsive pulp and apical breakdown.
Question 3
What is the diagnosis for tooth #4?
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Correct answer: B. Reversible pulpitis
- A. Asymptomatic irreversible pulpitis is diagnosed when there are no symptoms but the pulp is judged incapable of healing, often after caries excavation exposes the pulp.
- B. Correct. An exaggerated response that resolves promptly once the stimulus is removed is the definition of reversible pulpitis. Removing the irritant — here the deep restoration and any caries — should resolve it.
- C. A normal pulp gives a response proportional to the stimulus, not an exaggerated one.
- D. The absence of lingering is what separates this from irreversible pulpitis.
Question 4
Tooth #5 has pain on biting, percussion tenderness, and apical radiolucency. The periapical diagnosis is:
Show answer and explanation
Correct answer: A. Symptomatic apical periodontitis
- A. Correct. Pain to biting and percussion with apical radiographic change and no swelling or drainage defines symptomatic apical periodontitis.
- B. An acute apical abscess adds rapid onset, swelling, and often systemic signs such as fever and malaise.
- C. Condensing osteitis is a radiopaque, not radiolucent, response to low-grade pulpal irritation.
- D. A chronic apical abscess is characteristically asymptomatic and drains through a sinus tract.
Question 5
Which nerve fibers are responsible for the sensation elicited by an electric pulp test?
Show answer and explanation
Correct answer: C. A-delta fibers
- A. B fibers are preganglionic autonomic fibers and play no role in pulp testing.
- B. A-beta fibers carry touch and pressure from skin and are not the pulpal responders in an EPT.
- C. Correct. Myelinated A-delta fibers in the pulp–dentin border respond to the electrical stimulus and to cold, producing sharp, well-localized, brief pain.
- D. Unmyelinated C fibers are responsible for the dull, poorly localized, throbbing pain and for the response to heat — they are not activated by a standard EPT.
Question 6
Pain provoked by heat in a tooth with pulpal inflammation is carried predominantly by which fibers?
Show answer and explanation
Correct answer: D. C fibers
- A. A-alpha fibers are large motor and proprioceptive fibers, not pulpal nociceptors.
- B. Sympathetic fibers regulate pulpal blood flow rather than transmitting nociception.
- C. A-delta fibers mediate the sharp cold and EPT response, not the heat response.
- D. Correct. C fibers have a high threshold and are activated as inflammation and intrapulpal pressure rise, producing the dull, lingering, heat-provoked pain typical of advanced pulpitis.